Browse the latest research summaries in the field of pain management for spinal cord injury patients and caregivers.
Showing 311-320 of 555 results
Spinal Cord Series and Cases, 2017 • May 18, 2017
This correspondence critically evaluates a study comparing amitriptyline and lamotrigine for neuropathic pain after spinal cord injury. The authors highlight the scarcity of direct comparisons between...
KEY FINDING: The original study by Agarwal and Joshi reports the effectiveness of amitriptyline and lamotrigine in neuropathic pain after traumatic spinal cord injuries.
Neurobiol Dis, 2007 • June 1, 2007
This study investigates the role of spinal microglia in the anti-allodynic and anti-hyperalgesic effects of electroacupuncture (EA) in a rat model of monoarthritis induced by Complete Freund’s Adjuvan...
KEY FINDING: Unilateral intra-articular injection of CFA produced a robust microglial activation and the up-regulation of the tumor necrosis factor (TNF)-α, interleukin (IL-1β), and IL-6 mRNA levels in the spinal cord.
Pain, 2008 • July 31, 2008
This study investigated the role of TNF signaling in the development of CRPS-like changes in rats after tibia fracture. The researchers found that TNF expression was upregulated in the hindpaw skin an...
KEY FINDING: Tibia fracture upregulated TNF expression and protein levels in the hindpaw skin and sciatic nerve.
J Spinal Cord Med, 2007 • January 1, 2007
This review systematically examines outcome measures for pain after SCI, focusing on reliability, validity, sensitivity, and practicality to recommend specific measures for clinical trials. The review...
KEY FINDING: A 0–10 Point Numerical Rating Scale (NRS) is recommended as the outcome measure for pain intensity after SCI.
Spinal Cord, 2008 • June 1, 2008
This study examined the prevalence, intensity, and interference of pain in individuals with spinal cord injury (SCI). It investigated the relationship between pain characteristics and patient demograp...
KEY FINDING: Persons with higher level injuries were more likely to report upper extremity pain than persons with paraplegic injuries.
J Pain, 2008 • July 1, 2008
Results of this study provide additional preliminary support for the Motivational Model of Pain Self-Management. Contrary to our expectations, the motivational model could not be established for the c...
KEY FINDING: The effects of perceived importance and self-efficacy on exercise behavior were mediated by readiness to engage in exercise, consistent with the proposed model.
J Aging Health, 2008 • January 1, 2008
This study investigated age-related differences in pain coping strategies among adults with chronic pain secondary to disability. The study found that older adults used a wider range of coping strateg...
KEY FINDING: Older adults reported a wider range of frequently used strategies and significantly more frequent engagement in activity pacing, seeking social support, and use of coping self-statements than did younger or middle-aged adults.
Int J Clin Exp Hypn, 2009 • July 1, 2009
The study compared self-hypnosis (HYP) and EMG biofeedback (BIO) for chronic pain management in spinal cord injury (SCI) patients, finding both reduced immediate pain. HYP group showed lasting daily p...
KEY FINDING: Participants in both treatment conditions reported substantial, but similar, decreases in pain intensity from before to after the treatment sessions.
J Rehabil Res Dev, 2009 • January 1, 2009
This study replicated and extended previous research on psychosocial factors and chronic pain in individuals with SCI, using shortened measures of pain-related beliefs and coping. The results indicate...
KEY FINDING: Psychological factors, particularly beliefs about pain and pain-related coping strategies, were significant predictors of pain outcomes.
Pain, 2009 • October 1, 2009
The study investigated the analgesic effects of electroacupuncture (EA) on capsaicin-induced secondary hyperalgesia in rats, an animal model representing central sensitization. EA applied to specific ...
KEY FINDING: EA at acupoints SI3-TE8 significantly reduced capsaicin-induced secondary hyperalgesia, but not primary hyperalgesia.